Researchers at the University of California, San Diego School of Medicine say they have identified a set of laboratory-based biomarkers that can be useful for understanding brain-based abnormalities in schizophrenia. The measurements, known as endophenotypes, could ultimately be a boon to clinicians who sometimes struggle to recognize and treat the complex and confounding mental disorder.
Fudan University in Shanghai, China is partnering with the University of California to establish a Fudan-UC center to foster academic exchange and collaborative research on China. The center, a first-of-its-kind internationalization initiative made by a major Chinese university, will be located on the campus of the University of California, San Diego, hosted by the campus’ School of International Relations and Pacific Studies (IR/PS).
The biological mechanism of sunburn – the reddish, painful, protective immune response from ultraviolet (UV) radiation – is a consequence of RNA damage to skin cells, report researchers at the University of California, San Diego School of Medicine and elsewhere in the July 8, 2012 Advance Online Publication of Nature Medicine.
In the July 6 issue of Cell Stem Cell, researchers at the University of California, San Diego School of Medicine describe how human epidermal progenitor cells and stem cells control transcription factors to avoid premature differentiation, preserving their ability to produce new skin cells throughout life.
A new Web resource developed at the San Diego Supercomputer Center (SDSC) at the University of California, San Diego is helping thousands of researchers worldwide unravel the enigmas of phylogenetics, the study of evolutionary relationships among virtually every species on the planet.
Researchers at the University of California, San Diego – led by Gary S. Firestein, MD, professor in the Division of Rheumatology, Allergy and Immunology at UC San Diego School of Medicine – investigated a mechanism usually implicated in cancer and in fetal development, called DNA methylation, in the progression of rheumatoid arthritis (RA). They found that epigenetic changes due to methylation play a key role in altering genes that could potentially contribute to inflammation and joint damage.
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