Feb 24, 2015 San Diego, CA: Professors leading four new research centers at the University of California, San Diego Jacobs School of Engineering will speak at Research Expo on April 16, 2015. The faculty talks will focus on cutting-edge research in wearable sensors, extreme events research, sustainable power and energy, and visual computing. Learn more about the agile research centers project here.
Writing in the February 25 online issue of Nature, an international team of scientists, headed by researchers at the University of California, San Diego School of Medicine, report finding new links between inflammation and regeneration: signaling pathways that are activated by a receptor protein called gp130.
Writing in the February 25 online issue of the journal PLOS ONE, researchers at University of California, San Diego School of Medicine, with collaborators from The Scripps Research Institute (TSRI), have definitively shown for the first time that the culture conditions in which stem cells are grown and mass-produced can affect their genetic stability.
Late last year, two University of California, San Diego students set out for Florence, Italy, to diagnose a patient that had no prior medical record, couldn’t be poked or prodded in any way, and hadn’t been in prime condition for more than 800 years.
Chronic wasting disease (CWD) affects North American elk and deer, but has not been observed in humans. Using a mouse model that expresses an altered form of the normal human prion protein, researchers at University of California, San Diego School of Medicine have determined why the human proteins aren’t corrupted when exposed to the elk prions. Their study identifies a small loop in the human prion protein that confers resistance to chronic wasting disease.
Researchers at University of California, San Diego School of Medicine have discovered that the inflammatory molecule LTB4 promotes insulin resistance, a first step in developing type 2 diabetes. What’s more, the team found that genetically removing the cell receptor that responds to LTB4, or blocking it with a drug, improves insulin sensitivity in obese mice. The study is published Feb. 23 by Nature Medicine.
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