The Summer Institute is an all-expenses-paid, career-building fellowship for 25, first-year students from historically black colleges and universities and Hispanic-serving institutions across the U.S. The program is in part made possible by the partnership of corporate sponsors.
When University of California, San Diego alumnus and biotechnology pioneer David Goeddel, ’72, pledged a gift of $400,000 to establish the David V. Goeddel Endowed Graduate Fellowship at UC San Diego, his goal was to support and foster the innovators and scientists of the future. The endowed fund, which will be matched in full by the UC San Diego Division of Biological Sciences, will annually support the research and scholarly activities of outstanding Biological Sciences graduate students. The inaugural Goeddel fellowship recipients will use the funding to bolster leading-edge research focused on topics ranging from finding better treatments for diseases such as Parkinson’s, to understanding the metabolism of cells in tissues and organs, to studying cyanobacteria as a bio-production platform for renewable fuels.
The boxfish’s unique armor draws its strength from hexagon-shaped scales and the connections between them, engineers at the University of California, San Diego, have found. Engineers also describe how the structure of the boxfish (Lactoria cornuta) could serve as inspiration for body armor, robots and even flexible electronics.
Researchers at the University of California, San Diego School of Medicine have demonstrated a direct connection between two signaling proteins and liver fibrosis, a scarring process underlying chronic liver disease, the 12th leading cause of death in the United States.
Tapping the potential of metabolomics, an emerging field focused on the chemical processes of metabolism, researchers at University of California, San Diego School of Medicine have identified a new and pivotal player in diabetic kidney disease.
A new study by researchers at University of California, San Diego School of Medicine reveals a protein’s critical – and previously unknown – role in the development and progression of acute myeloid leukemia (AML), a fast-growing and extremely difficult-to-treat blood cancer. The finding offers a novel target for better treating AML, and possibly other cancers, by cutting off the ability of tumors to access nearby cellular players that feed its growth.
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