UC San Diego Sulpizio Cardiovascular Center has been named one of the nation’s 50 Top Cardiovascular Hospitals by Truven Health Analytics. The Truven study examined the performance of more than 1,000 hospitals by analyzing outcomes for patients with heart failure and heart attacks and for those who received coronary bypass surgery and percutaneous coronary interventions such as angioplasties.
Researchers at the University of California, San Diego School of Medicine report the discovery of microbial–dependent mechanisms through which some cancers mount an inflammatory response that fuels their development and growth.
The University of California, San Diego division of the California Institute for Telecommunications and Information Technology (Calit2) has given the green light to 17 new projects funded through the Calit2 Strategic Research Opportunities (CSRO) program. All told, the winning proposals garnered more than $826,000 in support from Calit2 for the year-long projects, effective October 1, 2012.
Two proteins previously found to contribute to ALS, also known as Lou Gehrig’s disease, have divergent roles. But a new study, led by researchers at the Department of Cellular and Molecular Medicine at the University of California, San Diego School of Medicine, shows that a common pathway links them.
An international consortium of scientists studying chronic lymphocytic leukemia (CLL), based at the University of California, San Diego School of Medicine, has been awarded a 5-year, $20 million grant by the National Cancer Institute, part of the National Institutes of Health. The grant is the second renewal of funding for a broad-based effort designed to better understand the pathology of CLL – the most common form of leukemia in the Western world – and develop new drugs and treatments.
Atherosclerosis has been presumed to be the consequence of complicated interactions between overabundant cholesterol and resulting inflammation in the heart and blood vessels. Researchers at the University of California, San Diego School of Medicine, with colleagues at institutions across the country, say the relationship is not exactly what it appears, and that a precursor to cholesterol actually suppresses inflammatory response genes. This precursor molecule could provide a new target for drugs designed to treat atherosclerosis.
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